Sepsis-associated acute kidney injury (SA-AKI) is frequently complicated by immune paralysis and kidney infection. Despite the spleen’s critical immunological role, the neural mechanisms regulating the splenic immune responses during SA-AKI remain unclear.
Kidney International published a clinical update in Research Highlights on 20 Feb 2026.
The item focuses on The splenic norepinephrine–β2-adrenergic receptor axis aggravates sepsis-associated acute kidney injury via neutrophil-mediated immunosuppression.
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